Control of erythroid cell production via caspase-mediated cleavage of transcription factor SCL/Tal-1
Open Access
- 17 July 2003
- journal article
- research article
- Published by Springer Nature in Cell Death & Differentiation
- Vol. 10 (8) , 905-913
- https://doi.org/10.1038/sj.cdd.4401255
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- New insights into erythropoiesisCurrent Opinion in Hematology, 2002
- Negative regulation of erythropoiesis by caspase-mediated cleavage of GATA-1Nature, 1999
- GATA-1 and Erythropoietin Cooperate to Promote Erythroid Cell Survival by Regulating bcl-xL ExpressionBlood, 1999
- bcl-x Prevents Apoptotic Cell Death of Both Primitive and Definitive Erythrocytes at the End of MaturationThe Journal of Experimental Medicine, 1999
- Apoptotic Role of Fas/Fas Ligand System in the Regulation of ErythropoiesisBlood, 1999
- Transcription factor GATA-1 permits survival and maturation of erythroid precursors by preventing apoptosis.Proceedings of the National Academy of Sciences, 1995
- Massive Cell Death of Immature Hematopoietic Cells and Neurons in Bcl-x-Deficient MiceScience, 1995
- Absence of blood formation in mice lacking the T-cell leukaemia oncoprotein tal-1/SCLNature, 1995
- The Oncogenic Cysteine-rich LIM domain protein Rbtn2 is essential for erythroid developmentCell, 1994
- bcl-x, a bcl-2-related gene that functions as a dominant regulator of apoptotic cell deathCell, 1993