Effect of Serotonin, Thromboxane A 2 , and Specific Receptor Antagonists on Vascular Smooth Muscle Cell Proliferation
- 7 October 1997
- journal article
- research article
- Published by Wolters Kluwer Health in Circulation
- Vol. 96 (7) , 2280-2286
- https://doi.org/10.1161/01.cir.96.7.2280
Abstract
Background Restenosis is a major complication that limits the long-term efficacy of coronary angioplasty. Migration and proliferation of activated medial smooth muscle cells (SMCs) is considered an...Keywords
This publication has 30 references indexed in Scilit:
- G proteinsPublished by Elsevier ,2003
- Local Effect of Serotonin Released during Coronary AngioplastyNew England Journal of Medicine, 1994
- Calcium Channels and Nifedipine Inhibition of Serotonin-Induced [3H]Thymidine Incorporation in Cultured Cerebral Smooth Muscle CellsJournal of Cerebral Blood Flow & Metabolism, 1992
- Syndromes of accelerated atherosclerosis: Role of vascular injury and smooth muscle cell proliferationJournal of the American College of Cardiology, 1990
- Mitogenic neurotransmittersNature, 1989
- Effect of 18- to 24-hour heparin administration for prevention of restenosis after uncomplicated coronary angioplastyAmerican Heart Journal, 1989
- Thromboxane and vascular smooth muscle cell growth in genetically hypertensive rats.Hypertension, 1988
- Aspirin and Dipyridamole in the Prevention of Re-Stenosis after Percutaneous Transluminal Coronary AngioplastyNew England Journal of Medicine, 1988
- The Pathogenesis of Atherosclerosis — An UpdateNew England Journal of Medicine, 1986
- Restenosis after percutaneous transluminal coronary angioplasty (PTCA): A report from the PTCA registry of the national heart, lung, and blood instituteThe American Journal of Cardiology, 1984