Human Papillomavirus Types 16 E6 and E7 Contribute Differently to Carcinogenesis
Top Cited Papers
- 1 February 2000
- Vol. 267 (2) , 141-150
- https://doi.org/10.1006/viro.1999.0106
Abstract
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This publication has 53 references indexed in Scilit:
- The link between integration and expression of human papillomavirus type 16 genomes and cellular changes in the evolution of cervical intraepithelial neoplastic lesions.Journal of General Virology, 1997
- Relating aromatic hydrocarbon-induced DNA adducts and c-H-ras mutations in mouse skin papillomas: the role of apurinic sites.Proceedings of the National Academy of Sciences, 1995
- Interaction of Papillomavirus E6 Oncoproteins with a Putative Calcium-Binding ProteinScience, 1995
- RAS Mutations in Human Melanoma: A Marker of Malignant ProgressionJournal of Investigative Dermatology, 1994
- Integration of human papillomavirus types 16 and 18 in cervical adenocarcinoma.Journal of Clinical Pathology, 1992
- Cigarette Smoking and the Risk of Anogenital CancerAmerican Journal of Epidemiology, 1992
- The Human Papilloma Virus-16 E7 Oncoprotein Is Able to Bind to the Retinoblastoma Gene ProductScience, 1989
- Human papillomavirus type 16 cooperates with activated ras and fos oncogenes in the hormone-dependent transformation of primary mouse cells.Proceedings of the National Academy of Sciences, 1988
- Integration of human papillomavirus type 16 into cellular DNA of cervical carcinoma: Preferential deletion of the E2 gene and invariable retention of the long control region and the E6/E7 open reading framesVirology, 1987
- v-ras genes from harvey and BALB murine sarcoma viruses can act as initiators of two-stage mouse skin carcinogenesisCell, 1986