The involvement of the interleukin-1 receptor-associated kinases (IRAKs) in cellular signaling networks controlling inflammation
- 30 April 2008
- Vol. 42 (1) , 1-7
- https://doi.org/10.1016/j.cyto.2007.12.012
Abstract
No abstract availableKeywords
This publication has 74 references indexed in Scilit:
- JNK1 in Hematopoietically Derived Cells Contributes to Diet-Induced Inflammation and Insulin Resistance without Affecting ObesityCell Metabolism, 2007
- Loss of the innate immunity negative regulator IRAK-M leads to enhanced host immune defense against tumor growthMolecular Immunology, 2007
- Essential role of IRAK-4 protein and its kinase activity in Toll-like receptor–mediated immune responses but not in TCR signalingThe Journal of Experimental Medicine, 2007
- Cross-talk between IRAK-4 and the NADPH oxidaseBiochemical Journal, 2007
- The association between innate immunity gene (IRAK1) and C-reactive protein in the Diabetes Heart StudyExperimental and Molecular Pathology, 2007
- Tumors induce a subset of inflammatory monocytes with immunosuppressive activity on CD8+ T cellsJournal of Clinical Investigation, 2006
- Sepsis-induced suppression of lung innate immunity is mediated by IRAK-MJournal of Clinical Investigation, 2006
- Autosomal recessive Interleukin-1 receptor-associated kinase 4 deficiency in fourth-degree relativesThe Journal of Pediatrics, 2006
- Critical role of TRAF3 in the Toll-like receptor-dependent and -independent antiviral responseNature, 2005
- Specificity in Toll-like receptor signalling through distinct effector functions of TRAF3 and TRAF6Nature, 2005