Thioredoxin suppresses airway hyperresponsiveness and airway inflammation in asthma
- 1 September 2005
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 334 (4) , 1141-1148
- https://doi.org/10.1016/j.bbrc.2005.07.007
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Culture, Sports, Science and Technology
This publication has 30 references indexed in Scilit:
- Enhanced Expression of Interleukin-18 and its Receptor in Idiopathic Pulmonary FibrosisAmerican Journal of Respiratory Cell and Molecular Biology, 2004
- Interleukin 18 (IL-18) in synergy with IL-2 induces lethal lung injury in mice: a potential role for cytokines, chemokines, and natural killer cells in the pathogenesis of interstitial pneumoniaBlood, 2002
- Circulating thioredoxin suppresses lipopolysaccharide-induced neutrophil chemotaxisProceedings of the National Academy of Sciences, 2001
- Interleukin-18 Regulates Both Th1 and Th2 ResponsesAnnual Review of Immunology, 2001
- The pharmacological basis of anti-IgE therapyNature Biotechnology, 2000
- Cytokines in asthmaThorax, 1999
- Difficult asthmaEuropean Respiratory Journal, 1998
- REDOX REGULATION OF CELLULAR ACTIVATIONAnnual Review of Immunology, 1997
- Redox control of resistance to cis-diamminedichloroplatinum (II) (CDDP): protective effect of human thioredoxin against CDDP-induced cytotoxicity.Journal of Clinical Investigation, 1996
- Attenuation of ischaemia reperfusion injury by human thioredoxin.Thorax, 1995