Integrin α1β1 and Transforming Growth Factor-β1 Play Distinct Roles in Alport Glomerular Pathogenesis and Serve as Dual Targets for Metabolic Therapy

This publication has 61 references indexed in Scilit:

• Insertional Mutation of the Collagen Genes Col4a3 and Col4a4 in a Mouse Model of Alport Syndrome
  Genomics, 1999
• Mesangial Cells and Their Adhesive Properties
  Nephron Experimental Nephrology, 1999
• Enhancement of Cell Adhesion and Spreading by a Cartilage-specific Noncollagenous Protein, Cartilage Matrix Protein (CMP/Matrilin-1), via Integrin α1β1
  Journal of Biological Chemistry, 1999
• The Laminin α2-Chain Short Arm Mediates Cell Adhesion through Both the α1β1 and α2β1 Integrins
  Journal of Biological Chemistry, 1997
• Alterations in the expression of the a 3 b 1 integrin in certain membrane domains of the glomerular epithelial cells (podocytes) in diabetes mellitus
  Diabetologia, 1997
• Molecular and functional defects in kidneys of mice lacking collagen alpha 3(IV): implications for Alport syndrome.
  The Journal of cell biology, 1996
• Transforming Growth Factor-β (TGF-β) Stimulates the Expression of β1 Integrins and Adhesion by Rat Mesangial Cells
  Experimental Cell Research, 1996
• Collagen IV alpha 3, alpha 4, and alpha 5 chains in rodent basal laminae: sequence, distribution, association with laminins, and developmental switches.
  The Journal of cell biology, 1994
• Mutations in the type IV collagen α3 (COL4A3) gene in autosomal recessive Alport syndrome
  Human Molecular Genetics, 1994
• Distribution of the α1 and α2 chains of collagen IV and of collagens V and VI in Alport syndrome
  Kidney International, 1992