Oral treatment of mice with copolymer 1 (glatiramer acetate) results in the accumulation of specific Th2 cells in the central nervous system
- 1 May 2002
- journal article
- Published by Elsevier in Journal of Neuroimmunology
- Vol. 126 (1-2) , 58-68
- https://doi.org/10.1016/s0165-5728(02)00053-x
Abstract
No abstract availableKeywords
This publication has 25 references indexed in Scilit:
- Glatiramer acetate (Copaxone®) induces degenerate, Th2-polarized immune responses in patients with multiple sclerosisJournal of Clinical Investigation, 2000
- The Fate of T Cells in the BrainThe American Journal of Pathology, 1998
- The central nervous system environment controls effector CD4+ T cell cytokine profile in experimental allergic encephalomyelitisEuropean Journal of Immunology, 1997
- Myelin Basic Protein–specific T Helper 2 (Th2) Cells Cause Experimental Autoimmune Encephalomyelitis in Immunodeficient Hosts Rather than Protect Them from the DiseaseThe Journal of Experimental Medicine, 1997
- Th1 and Th2 CD4+ T cells in the pathogenesis of organ-specific autoimmune diseasesImmunology Today, 1995
- Antigen-Specific Immunosuppression: Oral Tolerance for the Treatment of Autoimmune Disease (Part 1 of 2)Published by S. Karger AG ,1994
- T suppressor hybridomas and interleukin‐2‐dependent lines induced by copolymer 1 or by spinal cord homogenate down‐regulate experimental allergic encephalomyelitisEuropean Journal of Immunology, 1993
- Cellular immune reactivity within the CNSTrends in Neurosciences, 1986
- Suppression of experimental allergic encephalomyelitis by a synthetic polypeptideEuropean Journal of Immunology, 1971
- Basic encephalitogenic protein: A simplified purification on sulphoethyl‐sephadexFEBS Letters, 1970