Chapter 9 NMDA receptors and AMPA/kainate receptors mediate parallel injury in cerebral cortical cultures subjected to oxygen-glucose deprivation
- 1 January 1993
- book chapter
- Published by Elsevier
- Vol. 96, 137-143
- https://doi.org/10.1016/s0079-6123(08)63263-x
Abstract
No abstract availableThis publication has 28 references indexed in Scilit:
- Do NMDA Antagonists Prevent Neuronal Injury? Yes.Archives of Neurology, 1992
- Cerebral hypoxia: some new approaches and unanswered questionsJournal of Neuroscience, 1990
- Apparent hydroxyl radical production by peroxynitrite: implications for endothelial injury from nitric oxide and superoxide.Proceedings of the National Academy of Sciences, 1990
- Effect of zinc on NMDA receptor-mediated channel currents in cortical neuronsJournal of Neuroscience, 1990
- Direct Evidence That Excitotoxicity in Cultured Neurons Is Mediated via N‐Methyl‐D‐Aspartate (NMDA) as well as Non‐NMDA ReceptorsJournal of Neurochemistry, 1989
- Selective modulation of NMDA responses by reduction and oxidationNeuron, 1989
- Glutamate neurotoxicity and diseases of the nervous systemNeuron, 1988
- Mechanism of Kainate Toxicity to Cerebellar Neurons In Vitro Is Analogous to Reperfusion Tissue InjuryJournal of Neurochemistry, 1987
- Neurotoxicity of excitatory amino acid receptor agonists in rat cerebellar slices: Dependence on calcium concentrationNeuroscience Letters, 1986
- Cellular and molecular effects of polyunsaturated fatty acids in brain ischemia and injuryPublished by Elsevier ,1985