A Thyroid Hormone Receptor α Gene Mutation (P398H) Is Associated with Visceral Adiposity and Impaired Catecholamine-stimulated Lipolysis in Mice
Open Access
- 1 October 2003
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 278 (40) , 38913-38920
- https://doi.org/10.1074/jbc.m306120200
Abstract
No abstract availableKeywords
This publication has 55 references indexed in Scilit:
- Thyroid function and effect of aging in combined hetero/homozygous mice deficient in thyroid hormone receptors alpha and beta genesJournal of Endocrinology, 2002
- Sterol-dependent Transactivation of theABC1 Promoter by the Liver X Receptor/Retinoid X ReceptorJournal of Biological Chemistry, 2000
- Leptin, nutrition, and the thyroid: the why, the wherefore, and the wiringJournal of Clinical Investigation, 2000
- The central melanocortin system affects the hypothalamo-pituitary thyroid axis and may mediate the effect of leptinJournal of Clinical Investigation, 2000
- Rat MitochondrialGlycerol-3-Phosphate DehydrogenaseGene: Multiple Promoters, High Levels in Brown Adipose Tissue, and Tissue-Specific Regulation by Thyroid HormoneDNA and Cell Biology, 1998
- Thyroid hormone receptor β is essential for development of auditory functionNature Genetics, 1996
- α-AdrenoceptorsPharmacology & Therapeutics, 1994
- Lipolytic catecholamine resistance due to decreased beta 2-adrenoceptor expression in fat cells.Journal of Clinical Investigation, 1992
- Heterogeneous distribution of beta and alpha‐2 adrenoceptor binding sites in human fat cells from various fat deposits: functional consequencesEuropean Journal of Clinical Investigation, 1987
- Evidence that epinephrine acts preferentially as an antilipolytic agent in abdominal human subcutaneous fat cells: assessment by analysis of beta and alpha2 adrenoceptors propertiesEuropean Journal of Clinical Investigation, 1985