Deposition of Inhaled Pollen and Pollen Extract in Human Airways

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Abstract
IT is generally assumed that airborne grass-pollen grains are directly inhaled into the lungs and thereby lead to the development of the signs and symptoms of asthma in susceptible persons. This seemingly reasonable assumption, however, is confounded by aerodynamic calculations indicating that airborne particles of that size (20 to 30 μ in diameter) should not penetrate very deeply into the respiratory system.1 In addition, since aerodynamic calculations are basically statistical descriptions, there is some uncertainty about possible deep penetration of occasional pollen grains into the lung, with subsequent symptom production. Because of the importance of this problem and the availability . . .