Inflammation and drug hepatotoxicity

This publication has 17 references indexed in Scilit:

• Mitochondrial permeability transition in acetaminophen-induced necrosis and apoptosis of cultured mouse hepatocytes
  Hepatology, 2004
• Reduced oncotic necrosis in fas receptor-deficient C57BL/6J- lpr mice after bile duct ligation
  Hepatology, 2004
• Acetaminophen and the U.S. acute liver failure study group: Lowering the risks of hepatic failure
  Hepatology, 2004
• Apoptosis and necrosis in liver disease
  Liver International, 2004
• Acetaminophen-Induced Oxidant Stress and Cell Injury in Cultured Mouse Hepatocytes: Protection by N-Acetyl Cysteine
  Toxicological Sciences, 2004
• Subliminal Fas stimulation increases the hepatotoxicity of acetaminophen and bromobenzene in mice
  Hepatology, 2004
• Acetaminophen-Induced Inhibition of Fas Receptor-Mediated Liver Cell Apoptosis: Mitochondrial Dysfunction versus Glutathione Depletion
  Toxicology and Applied Pharmacology, 2002
• Pretreatment of mice with macrophage inactivators decreases acetaminophen hepatotoxicity and the formation of reactive oxygen and nitrogen species
  Hepatology, 1999
• The development of autoimmunity in C57BL/6 lpr mice correlates with the disappearance of natural killer type 1-positive cells: evidence for their suppressive action on bone marrow stem cell proliferation, B cell immunoglobulin secretion, and autoimmune symptoms.
  The Journal of Experimental Medicine, 1993
• Molecular Mechanisms of the Hepatotoxicity Caused by Acetaminophen
  Seminars in Liver Disease, 1990