The IKK-2/IκBα/NF-κB Pathway Plays a Key Role in the Regulation of CCR3 and eotaxin-1 in Fibroblasts

This publication has 37 references indexed in Scilit:

• Cell-Type-Dependent Induction of eotaxin and CCR3 by Ionizing Radiation
  Biochemical and Biophysical Research Communications, 2000
• Mice lacking the transcription factor RelB develop T cell-dependent skin lesions similar to human atopic dermatitis
  European Journal of Immunology, 2000
• Activators and target genes of Rel/NF-κB transcription factors
  Oncogene, 1999
• Chemokines and asthma: redundancy of function or a coordinated effort?
  Journal of Clinical Investigation, 1999
• Genomic Organization, Sequence, and Transcriptional Regulation of the Human Eotaxin Gene
  Biochemical and Biophysical Research Communications, 1997
• Genomic Organization, Complete Sequence, and Chromosomal Location of the Gene for Human Eotaxin (SCYA11), an Eosinophil-Specific CC Chemokine
  Genomics, 1997
• Human Chemokines: An Update
  Annual Review of Immunology, 1997
• Human Dermal Fibroblasts Express Eotaxin: Molecular Cloning, mRNA Expression, and Identification of Eotaxin Sequence Variants
  Biochemical and Biophysical Research Communications, 1996
• Differential in situ cytokine gene expression in acute versus chronic atopic dermatitis.
  Journal of Clinical Investigation, 1994
• The Chemokine, Eotaxin, Activates Guinea-Pig Eosinophils in Vitro and Causes Their Accumulation into the Lung in Vivo
  Biochemical and Biophysical Research Communications, 1993