Collapse of the Inner Mitochondrial Transmembrane Potential Is Not Required for Apoptosis of HL60 Cells
- 1 August 1999
- journal article
- Published by Elsevier in Experimental Cell Research
- Vol. 251 (1) , 166-174
- https://doi.org/10.1006/excr.1999.4527
Abstract
No abstract availableKeywords
This publication has 77 references indexed in Scilit:
- Oxidative stress as a mediator of apoptosisPublished by Elsevier ,2002
- Release of Cytochrome c from Liver Mitochondria during Permeability TransitionBiochemical and Biophysical Research Communications, 1997
- Bcl-xL forms an ion channel in synthetic lipid membranesNature, 1997
- ICE Family Proteases: Mediators of All Apoptotic Cell Death?Immunity, 1996
- Inhibition of the Caenorhabditis elegans cell-death protease CED-3 by a CED-3 cleavage site in baculovirus p35 proteinNature, 1995
- The Baculovirus p35 Protein Inhibits Fas- and Tumor Necrosis Factor-induced ApoptosisPublished by Elsevier ,1995
- Alterations in mitochondrial structure and function are early events of dexamethasone-induced thymocyte apoptosis.The Journal of cell biology, 1995
- The mitochondrial permeability transitionBiochimica et Biophysica Acta (BBA) - Reviews on Biomembranes, 1995
- No requirement of reactive oxygen intermediates in Fas‐mediated apoptosisFEBS Letters, 1994
- Bcl-2 is an inner mitochondrial membrane protein that blocks programmed cell deathNature, 1990