Caspase-9 Activation and Caspase Cleavage of tau in the Alzheimer's Disease Brain
- 1 November 2002
- journal article
- Published by Elsevier in Neurobiology of Disease
- Vol. 11 (2) , 341-354
- https://doi.org/10.1006/nbdi.2002.0549
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- The Role of Oxidative Stress in the Pathophysiology of Cerebrovascular Lesions in Alzheimer's DiseaseBrain Pathology, 2002
- Proapoptotic Effects of Tau Cleavage Product Generated by Caspase-3Neurobiology of Disease, 2001
- The Neuronal Microtubule‐Associated Protein Tau Is a Substrate for Caspase‐3 and an Effector of ApoptosisJournal of Neurochemistry, 2000
- Tau protein isoforms, phosphorylation and role in neurodegenerative disorders11These authors contributed equally to this work.Brain Research Reviews, 2000
- Apoptosis control by death and decoy receptorsPublished by Elsevier ,1999
- Involvement of Caspases in Proteolytic Cleavage of Alzheimer’s Amyloid-β Precursor Protein and Amyloidogenic Aβ Peptide FormationCell, 1999
- Tau Cleavage and Dephosphorylation in Cerebellar Granule Neurons Undergoing ApoptosisJournal of Neuroscience, 1998
- Alzheimer's disease hyperphosphorylated tau sequesters normal tau into tangles of filaments and disassembles microtubulesNature Medicine, 1996
- β-Amyloid Accumulation Correlates with Cognitive Dysfunction in the Aged CanineNeurobiology of Learning and Memory, 1996
- Abnormal tau phosphorylation at Ser396 in alzheimer's disease recapitulates development and contributes to reduced microtubule bindingNeuron, 1993