Gelatinase B (MMP-9) Is Not Essential in the Normal Kidney and Does Not Influence Progression of Renal Disease in a Mouse Model of Alport Syndrome
Open Access
- 31 July 2000
- journal article
- Published by Elsevier in The American Journal of Pathology
- Vol. 157 (1) , 303-311
- https://doi.org/10.1016/s0002-9440(10)64541-5
Abstract
No abstract availableKeywords
This publication has 45 references indexed in Scilit:
- Gelatinase B–deficient Mice Are Resistant to Experimental Bullous PemphigoidThe Journal of Experimental Medicine, 1998
- Differential expression of five laminin α (1-5) chains in developing and adult mouse kidneyDevelopmental Dynamics, 1997
- Mighty mice: Transgenic technology “knocks out” questions of matrix metalloproteinase functionMatrix Biology, 1997
- Molecular and functional defects in kidneys of mice lacking collagen alpha 3(IV): implications for Alport syndrome.The Journal of cell biology, 1996
- Matrix metalloproteinases and TIMPS in cultured C57BL/6J-cpk kidney tubulesKidney International, 1996
- Developmental genetics of the heartCurrent Opinion in Genetics & Development, 1996
- Collagen IV alpha 3, alpha 4, and alpha 5 chains in rodent basal laminae: sequence, distribution, association with laminins, and developmental switches.The Journal of cell biology, 1994
- Tumor Cell Interactions with the Extracellular Matrix During Invasion and MetastasisAnnual Review of Cell Biology, 1993
- Loss of laminin epitopes during glomerular basement membrane assembly in developing mouse kidneys.Journal of Histochemistry & Cytochemistry, 1992
- Development of tubular and glomerular cells of the kidneyKidney International, 1992