Apoptosis and JNK activation are differentially regulated by Fas expression level in renal tubular epithelial cells
- 1 July 2001
- journal article
- Published by Elsevier in Kidney International
- Vol. 60 (1) , 65-76
- https://doi.org/10.1046/j.1523-1755.2001.00771.x
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- Signal transduction by the c-Jun N-terminal kinase (JNK) — from inflammation to developmentCurrent Opinion in Cell Biology, 1998
- Early Activation of c-Jun N-terminal Kinase and p38 Kinase Regulate Cell Survival in Response to Tumor Necrosis Factor αJournal of Biological Chemistry, 1998
- MAP3K-related kinase involved in NF-KB induction by TNF, CD95 and IL-1Nature, 1997
- Apoptosis by Death FactorCell, 1997
- Stress-signalling kinase Sek1 protects thymocytes from apoptosis mediated by CD95 and CD3Nature, 1997
- Dissection of TNF Receptor 1 Effector Functions: JNK Activation Is Not Linked to Apoptosis While NF-κB Activation Prevents Cell DeathCell, 1996
- Fas Ligand-Induced Apoptosis as a Mechanism of Immune PrivilegeScience, 1995
- Protection from Fas-Mediated Apoptosis by a Soluble Form of the Fas MoleculeScience, 1994
- JNK1: A protein kinase stimulated by UV light and Ha-Ras that binds and phosphorylates the c-Jun activation domainPublished by Elsevier ,1994
- Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosisNature, 1992