SOCS3 Is Essential in the Regulation of Fetal Liver Erythropoiesis
- 1 September 1999
- Vol. 98 (5) , 617-627
- https://doi.org/10.1016/s0092-8674(00)80049-5
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Fetal Anemia and Apoptosis of Red Cell Progenitors in Stat5a−/−5b−/− MiceCell, 1999
- The Elongin BC complex interacts with the conserved SOCS-box motif present in members of the SOCS, ras, WD-40 repeat, and ankyrin repeat familiesGenes & Development, 1998
- FADD: Essential for Embryo Development and Signaling from Some, But Not All, Inducers of ApoptosisScience, 1998
- Identification of SOCS-3 as a Potential Mediator of Central Leptin ResistanceMolecular Cell, 1998
- Bcl-2 Rescues T Lymphopoiesis, but Not B or NK Cell Development, in Common γ Chain–Deficient MiceImmunity, 1997
- DNA ligase I is required for fetal liver erythropoiesis but is not essential for mammalian cell viabilityNature Genetics, 1996
- Generation of committed erythroid BFU-E and CFU-E progenitors does not require erythropoietin or the erythropoietin receptorCell, 1995
- Interaction of the erythropoietin and stem-cell-factor receptorsNature, 1995
- Specific recruitment of SH-PTP1 to the erythropoietin receptor causes inactivation of JAK2 and termination of proliferative signalsCell, 1995
- A developmental switch in thymic lymphocyte maturation potential occurs at the level of hematopoietic stem cellsCell, 1990