Postprandial glucagon-like peptide-1 (GLP-1) response is positively associated with changes in neuronal activity of brain areas implicated in satiety and food intake regulation in humans

This publication has 42 references indexed in Scilit:

• In pursuit of neural risk factors for weight gain in humans
  Neurobiology of Aging, 2005
• The expression of GLP‐1 receptor mRNA and protein allows the effect of GLP‐1 on glucose metabolism in the human hypothalamus and brainstem
  Journal of Neurochemistry, 2005
• Neuroimaging and Obesity
  Annals of the New York Academy of Sciences, 2002
• Classical and Bayesian Inference in Neuroimaging: Applications
  NeuroImage, 2002
• Classical and Bayesian Inference in Neuroimaging: Theory
  NeuroImage, 2002
• The role of postprandial releases of insulin and incretin hormones in meal-induced satiety—effect of obesity and weight reduction
  International Journal of Obesity, 2001
• The effect of physiological levels of glucagon-like peptide-1 on appetite, gastric emptying, energy and substrate metabolism in obesity
  International Journal of Obesity, 2001
• Hunger selectively modulates corticolimbic activation to food stimuli in humans.
  Behavioral Neuroscience, 2001
• The reproducibility of subjective appetite scores
  British Journal of Nutrition, 1995
• Tissue‐specific expression of the human receptor for glucagon‐like peptide‐I: brain, heart and pancreatic forms have the same deduced amino acid sequences
  FEBS Letters, 1995