Annexin II and Bleeding in Acute Promyelocytic Leukemia
Open Access
- 1 April 1999
- journal article
- research article
- Published by Massachusetts Medical Society in New England Journal of Medicine
- Vol. 340 (13) , 994-1004
- https://doi.org/10.1056/nejm199904013401303
Abstract
Acute promyelocytic leukemia (APL) is associated with a hemorrhagic disorder of unknown cause that responds to treatment with all-trans-retinoic acid.Keywords
This publication has 52 references indexed in Scilit:
- Interaction of the Fibrinolytic Receptor, Annexin II, with the Endothelial Cell SurfaceJournal of Biological Chemistry, 1996
- Thrombosis in patients with acute promyelocytic leukemia treated with and without all-trans retinoic acidLeukemia & Lymphoma, 1996
- Four years' experience with the treatment of all‐trans retinoic acid in acute promyelocytic leukemiaAmerican Journal of Hematology, 1993
- Successful Treatment with Tranexamic Acid for Severe Bleeding in Acute Promyelocytic LeukemiaActa Haematologica, 1992
- The bleeding disorder in acute promyelocytic leukaemia: fibrinolysis due to u‐PA rather than defibrinationBritish Journal of Haematology, 1989
- Stimulation of 3T3 cells induces transcription of the c-fos proto-oncogeneNature, 1984
- The behavior of alpha2-plasmin inhibitor in fibrinolytic states.Journal of Clinical Investigation, 1977
- Platelet and fibrinogen survival in acute promyelocytic leukaemia.BMJ, 1976
- ε-Amino-Caproic Acid in the Management of Acute Promyelocytic LeukaemiaActa Haematologica, 1976
- Fibrinolysis in leukaemiaJournal of Clinical Pathology, 1968