Theoretical Analysis of the Implication of PrP in Neuronal Death during Transmissible Subacute Spongiform Encephalopathies: Hypothesis of a PrP Oligomeric Channel
- 7 May 2000
- journal article
- Published by Elsevier in Journal of Theoretical Biology
- Vol. 204 (1) , 103-111
- https://doi.org/10.1006/jtbi.2000.1091
Abstract
No abstract availableKeywords
This publication has 50 references indexed in Scilit:
- The 118–135 peptide of the human prion protein forms amyloid fibrils and induces liposome fusionJournal of Molecular Biology, 1997
- A Neurotoxic and Gliotrophic Fragment of the Prion Protein Increases Plasma Membrane MicroviscosityNeurobiology of Disease, 1997
- Annexins taken to taskNature, 1995
- Mouse cortical cells lacking cellular PrP survive in culture with a neurotoxic PrP fragmentNeuroReport, 1994
- A Neurotoxic Prion Protein Fragment Induces Rat Astroglial Proliferation and HypertrophyEuropean Journal of Neuroscience, 1994
- Cytoprotective effect of NMDA receptor antagonists on prion protein (PrionSc)-induced toxicity in rat cortical cell culturesEuropean Journal of Pharmacology: Molecular Pharmacology, 1993
- Crystal and molecular structure of human annexin V after refinement: Implications for structure, membrane binding and ion channel formation of the annexin family of proteinsJournal of Molecular Biology, 1992
- Pro→Leu change at position 102 of prinon protein is the most common but not the sole mutation related to Gerstmann-Sträussler syndromeBiochemical and Biophysical Research Communications, 1989
- Unraveling prion diseases through molecular geneticsTrends in Neurosciences, 1989
- CHARMM: A program for macromolecular energy, minimization, and dynamics calculationsJournal of Computational Chemistry, 1983