KAL, a gene mutated in Kallmann's syndrome, is expressed in the first trimester of human development
- 28 April 1995
- journal article
- Published by Elsevier in Molecular and Cellular Endocrinology
- Vol. 110 (1-2) , 73-79
- https://doi.org/10.1016/0303-7207(95)03518-c
Abstract
No abstract availableKeywords
This publication has 13 references indexed in Scilit:
- Expression of the Kallmann syndrome gene in human fetal brain and in the manipulated chick embryoHuman Molecular Genetics, 1994
- Unilateral renal aplasia in X‐linked Kallmann's syndromeClinical Genetics, 1994
- Retinal signal transmission in Duchenne muscular dystrophy: evidence for dysfunction in the photoreceptor/depolarizing bipolar cell pathway.Journal of Clinical Investigation, 1994
- Expression pattern of the Kallmann syndrome gene in the olfactory system suggests a role in neuronal targetingNature Genetics, 1993
- Xp22.3 deletions in isolated familial Kallmann's syndromeJournal of Clinical Endocrinology & Metabolism, 1993
- Sex and smell – an enigma resolvedJournal of Endocrinology, 1992
- The candidate gene for the X-linked Kallmann syndrome encodes a protein related to adhesion molecules.Published by Elsevier ,1991
- A gene deleted in Kallmann's syndrome shares homology with neural cell adhesion and axonal path-finding moleculesNature, 1991
- Luteinizing hormone-releasing hormone (LHRH)-expressing cells do not migrate normally in an inherited hypogonadal (Kallmann) syndromeMolecular Brain Research, 1989
- Role of laminin a chain in the development of epithelial cell polarityCell, 1988