Manganese Superoxide Dismutase Protects Mitochondrial Complex I against Adriamycin-Induced Cardiomyopathy in Transgenic Mice
- 1 February 1999
- journal article
- Published by Elsevier in Archives of Biochemistry and Biophysics
- Vol. 362 (1) , 59-66
- https://doi.org/10.1006/abbi.1998.1011
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- Excessive formation of hydroxyl radicals and aldehydic lipid peroxidation products in cultured skin fibroblasts from patients with complex I deficiency.Journal of Clinical Investigation, 1997
- Nitric oxide, superoxide, and peroxynitrite: the good, the bad, and uglyAmerican Journal of Physiology-Cell Physiology, 1996
- Mitochondrial complex I deficiency leads to increased production of superoxide radicals and induction of superoxide dismutase.Journal of Clinical Investigation, 1996
- SUPEROXIDE RADICAL AND SUPEROXIDE DISMUTASESAnnual Review of Biochemistry, 1995
- An analysis of the role of coenzyme Q in free radical generation and as an antioxidantBiochemistry and Cell Biology, 1992
- The molecular pharmacology of doxorubicin in vivoEuropean Journal of Cancer and Clinical Oncology, 1991
- Free radical formation by antitumor quinonesFree Radical Biology & Medicine, 1989
- Subcellular effects of adriamycin in the heart: A concise reviewJournal of Molecular and Cellular Cardiology, 1987
- THE MITOCHONDRIAL ELECTRON TRANSPORT AND OXIDATIVE PHOSPHORYLATION SYSTEMAnnual Review of Biochemistry, 1985
- Mitochondrial Superoxide DismutaseJournal of Biological Chemistry, 1973