CCL20/macrophage inflammatory protein-3? production in LPS-stimulated neutrophils is enhanced by the chemoattractant formyl-methionyl-leucyl-phenylalanine and IFN-? through independent mechanisms
Open Access
- 19 November 2002
- journal article
- Published by Wiley in European Journal of Immunology
- Vol. 32 (12) , 3515-3524
- https://doi.org/10.1002/1521-4141(200212)32:12<3515::aid-immu3515>3.0.co;2-3
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- Human T cell leukemia virus type-I Tax activates human macrophage inflammatory protein-3α/CCL20 gene transcription via the NF-κB pathwayInternational Immunology, 2002
- Proinflammatory cytokines induce liver and activation-regulated chemokine/macrophage inflammatory protein-3α/CCL20 in mucosal epithelial cells through NF-κBInternational Immunology, 2001
- The neutrophil as a cellular source of chemokinesImmunological Reviews, 2000
- Modulation by Interferon-γ of the Production and Gene Expression of IL-1 Receptor Antagonist in Human NeutrophilsCellular Immunology, 1998
- The production of cytokines by polymorphonuclear neutrophilsImmunology Today, 1995
- Modulation of Proinflammatory Cytokine Release from Human Polymorphonuclear Leukocytes by Gamma InterferonCellular Immunology, 1994
- Rapid dendritic cell recruitment is a hallmark of the acute inflammatory response at mucosal surfaces.The Journal of Experimental Medicine, 1994
- Interleukin 10 (IL-10) inhibits the release of proinflammatory cytokines from human polymorphonuclear leukocytes. Evidence for an autocrine role of tumor necrosis factor and IL-1 beta in mediating the production of IL-8 triggered by lipopolysaccharide.The Journal of Experimental Medicine, 1993
- Lipopolysaccharide priming of human neutrophils for an enhanced respiratory burst. Role of intracellular free calcium.Journal of Clinical Investigation, 1989
- Priming of neutrophils for enhanced release of oxygen metabolites by bacterial lipopolysaccharide. Evidence for increased activity of the superoxide-producing enzyme.The Journal of Experimental Medicine, 1984