Radiosensitization of hypoxic tumour cells by S-nitroso-N-acetylpenicillamine implicates a bioreductive mechanism of nitric oxide generation
Open Access
- 12 February 1999
- journal article
- Published by Springer Nature in British Journal of Cancer
- Vol. 79 (7) , 1085-1089
- https://doi.org/10.1038/sj.bjc.6690173
Abstract
No abstract availableKeywords
This publication has 12 references indexed in Scilit:
- Intrinsic radiosensitivity of human pancreatic tumour cells and the radiosensitising potency of the nitric oxide donor sodium nitroprussideBritish Journal of Cancer, 1996
- Enhanced release of nitric oxide causes increased cytotoxicity of S-nitroso-N-acetyl-dl-penicillamine and sodium nitroprusside under hypoxic conditionsBiochemical Journal, 1996
- Bioactivation of Nitroprusside by Porcine Endothelial CellsToxicology and Applied Pharmacology, 1994
- Nitric oxide reacts with intracellular glutathione and activates the hexose monophosphate shunt in human neutrophils: evidence for S-nitrosoglutathione as a bioactive intermediary.Proceedings of the National Academy of Sciences, 1994
- Hypoxic mammalian cell radiosensitization by nitric oxide.1993
- Biochemistry of Nitric Oxide and Its Redox-Activated FormsScience, 1992
- Failla Memorial Lecture. Redox, radiation, and reductive bioactivation.1992
- Redox, Radiation, and Reductive BioactivationRadiation Research, 1992
- Nitric oxide generation from nitroprusside by vascular tissueBiochemical Pharmacology, 1991
- Effect of Nitric Oxide on the Radiosensitivity of BacteriaNature, 1957