Drug Sensitivity in Hereditary Hepatic Porphyria
- 1 January 1978
- book chapter
- Published by Springer Nature
- No. 1,p. 71-78
- https://doi.org/10.1007/978-3-642-67179-1_11
Abstract
No abstract availableKeywords
This publication has 11 references indexed in Scilit:
- Reduced Ferrochelatase Activity: a Defect Common to Porphyria Variegata and ProtoporphyriaBritish Journal of Haematology, 1977
- Decreased lymphocyte coproporphyrinogen III oxidase activity in hereditary coproporphyriaBiochemical and Biophysical Research Communications, 1977
- THE PRIMARY ENZYME DEFECT IN HEREDITARY COPROPORPHYRIAThe Lancet, 1976
- Effect of Lead on Hepatic δ-Aminolaevulinic Acid Synthetase Activity in the Rat: A Model for Drug Sensitivity in Intermittent Acute PorphyriaEuropean Journal of Clinical Investigation, 1976
- Studies in porphyria; v. Drug oxidation rates in hereditary hepatic porphyriaClinical Pharmacology & Therapeutics, 1976
- EFFECT OF LEAD AND GENETIC FACTORS ON HEME BIOSYNTHESIS IN THE HUMAN RED CELL*Annals of the New York Academy of Sciences, 1975
- Salicylamide metabolism in acute intermittent porphyriaClinical Pharmacology & Therapeutics, 1974
- Intermittent Acute PorphyriaEnzyme, 1973
- Intermittent Acute Porphyria — Demonstration of a Genetic Defect in Porphobilinogen MetabolismNew England Journal of Medicine, 1972
- Radiochemical microassay of δ-aminolevulinic acid synthetase in hepatic and erythroid tissuesAnalytical Biochemistry, 1972