Degradation of p53 only is not sufficient for the growth stimulatory effect of human papillomavirus 16 E6 oncoprotein in human embryonic fibroblasts
- 1 November 1994
- journal article
- research article
- Published by Wiley in Journal of Medical Virology
- Vol. 44 (3) , 243-249
- https://doi.org/10.1002/jmv.1890440306
Abstract
No abstract availableKeywords
This publication has 27 references indexed in Scilit:
- Degradation of p53 can be targeted by HPV E6 sequences distinct from those required for p53 binding and trans-activationCell, 1991
- Cooperative effect of antisense-Rb and antisense-p53 oligomers on the extension of life span in human diploid fibroblasts, TIG-1Biochemical and Biophysical Research Communications, 1991
- Wild-type p53 can inhibit oncogene-mediated focus formation.Proceedings of the National Academy of Sciences, 1989
- The p53 proto-oncogene can act as a suppressor of transformationCell, 1989
- The Human Papilloma Virus-16 E7 Oncoprotein Is Able to Bind to the Retinoblastoma Gene ProductScience, 1989
- Human papillomavirus type 16 cooperates with activated ras and fos oncogenes in the hormone-dependent transformation of primary mouse cells.Proceedings of the National Academy of Sciences, 1988
- Redesign of retrovirus packaging cell lines to avoid recombination leading to helper virus production.Molecular and Cellular Biology, 1986
- A papillomavirus DNA from a cervical carcinoma and its prevalence in cancer biopsy samples from different geographic regions.Proceedings of the National Academy of Sciences, 1983
- Isolation of biologically active ribonucleic acid from sources enriched in ribonucleaseBiochemistry, 1979
- Polyadenylic Acid Sequences in the Heterogeneous Nuclear RNA and Rapidly-Labeled Polyribosomal RNA of HeLa Cells: Possible Evidence for a Precursor RelationshipProceedings of the National Academy of Sciences, 1971