Endogenous AP-1 Levels Necessary for Oncogenic Activity Are Higher Than Those Sufficient to Support Normal Growth
- 11 November 2000
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 278 (1) , 97-105
- https://doi.org/10.1006/bbrc.2000.3777
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- Retrovirus vectors designed for efficient transduction of cytotoxic or cytostatic genesGene Therapy, 1999
- Two Proteins Translated by Alternative Usage of Initiation Codons in mRNA Encoding a JunD Transcriptional RegulatorBiochemical and Biophysical Research Communications, 1998
- Phosphorylation and high level expression of Fra-2 in v-src transformed cells: a pathway of activation of endogenous AP-1Oncogene, 1997
- Transformation by ras modifies AP1 composition and activityOncogene, 1997
- Activation and inhibition of the AP-1 complex in human breast cancer cellsMolecular Carcinogenesis, 1996
- Activation of an inducible c-FosER fusion protein causes loss of epithelial polarity and triggers epithelial-fibroblastoid cell conversionCell, 1992
- The role of Jun, Fos and the AP-1 complex in cell-proliferation and transformationBiochimica et Biophysica Acta (BBA) - Reviews on Cancer, 1991
- Transformation suppressor activity of a Jun transcription factor lacking its activation domainNature, 1991
- High‐level Expression of Human c‐jun Gene Causes Cellular Transformation of Chicken Embryo FibroblastsJapanese Journal of Cancer Research, 1991
- Phorbol ester-inducible genes contain a common cis element recognized by a TPA-modulated trans-acting factorCell, 1987