A Role for Tumor Necrosis Factor Receptor-2 and Receptor-interacting Protein in Programmed Necrosis and Antiviral Responses
Open Access
- 1 December 2003
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 278 (51) , 51613-51621
- https://doi.org/10.1074/jbc.m305633200
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- Caspase-independent cell death in T lymphocytesNature Immunology, 2003
- Bacterial strategies for overcoming host innate and adaptive immune responsesNature Immunology, 2002
- The Death Domain Kinase RIP Protects Thymocytes from Tumor Necrosis Factor Receptor Type 2–induced Cell DeathThe Journal of Experimental Medicine, 2002
- The Danger Model: A Renewed Sense of SelfScience, 2002
- RICK/Rip2/CARDIAK mediates signalling for receptors of the innate and adaptive immune systemsNature, 2002
- Involvement of receptor-interacting protein 2 in innate and adaptive immune responsesNature, 2002
- Immunology 102 at poxvirus U: Avoiding apoptosisSeminars in Immunology, 2001
- Resistance to the Cytotoxic Effects of Tumor Necrosis Factor α Can Be Overcome by Inhibition of a FADD/Caspase-dependent Signaling PathwayPublished by Elsevier ,1999
- TUMOR NECROSIS FACTOR RECEPTOR AND Fas SIGNALING MECHANISMSAnnual Review of Immunology, 1999
- RIP: A novel protein containing a death domain that interacts with Fas/APO-1 (CD95) in yeast and causes cell deathCell, 1995