Defects in Optokinetic Afternystagmus in Lesions of the Brain Stem

Abstract
Optokinetic afternystagmus (OKAN) was investigated in 50 rhesus monkeys. All had electrolytic lesions in the brain stem. Twenty-two of them showed a unilateral defect in OKAN. This defect was correlated with the location of the lesion in the oculomotor pathways. Twenty-eight monkeys showed no such defect. In absence of other measurable signs OKAN can serve as a sensitive index of a type of oculomotor dysfunction.

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