High glucose alters the response of mesangial cell protein kinase C isoforms to endothelin-1
- 1 February 1999
- journal article
- Published by Elsevier in Kidney International
- Vol. 55 (2) , 486-499
- https://doi.org/10.1046/j.1523-1755.1999.00284.x
Abstract
No abstract availableKeywords
This publication has 41 references indexed in Scilit:
- Effect of a Specific Endothelin Receptor A Antagonist on mRNA Levels for Extracellular Matrix Components and Growth Factors in Diabetic GlomeruliDiabetes, 1995
- Expression of growth-related protooncogenes during diabetic renal hypertrophyKidney International, 1995
- Platelet-derived growth factor and angiotensin II stimulate the mitogen-activated protein kinase cascade in renal mesangial cells: comparison of hypertrophic and hyperplastic agonistsBiochemical Journal, 1995
- High glucose elevates c-fos and c-jun transcripts and proteins in mesangial cell culturesKidney International, 1994
- Platelet-derived growth factor and angiotensin II induce different spatial distribution of protein kinase C-alpha and -beta in vascular smooth muscle cells.Hypertension, 1994
- Role for Protein Kinase C in the Mediation of Increased Fibronectin Accumulation by Mesangial Cells Grown in High-Glucose MediumDiabetes, 1993
- Receptor-specific increase in extracellular matrix production in mouse mesangial cells by advanced glycosylation end products is mediated via platelet-derived growth factor.Proceedings of the National Academy of Sciences, 1992
- Protein Kinase C Regulates Activation of Mitogen-Activated Protein Kinase and Induction of Proto-oncogene c-fos by Endothelin-1Journal of Cardiovascular Pharmacology, 1992
- Diabetes-induced glomerular dysfunction: Links to a more reduced cytosolic ratio of NADH/NAD+Kidney International, 1992
- Protein kinase C is activated in glomeruli from streptozotocin diabetic rats. Possible mediation by glucose.Journal of Clinical Investigation, 1989