Differential regulation of Rb family proteins and prohibitin during camptothecin-induced apoptosis
- 27 June 2002
- journal article
- Published by Springer Nature in Oncogene
- Vol. 21 (29) , 4539-4548
- https://doi.org/10.1038/sj.onc.1205551
Abstract
Oncogene is one of the world’s leading cancer journals. It is published weekly and covers all aspects of the structure and function of Oncogenes. Oncogene also publishes 8 Reviews issues a year, on a broad range of topics.Keywords
This publication has 39 references indexed in Scilit:
- E2Fs regulate the expression of genes involved in differentiation, development, proliferation, and apoptosisGenes & Development, 2001
- Prohibitin, a potential tumor suppressor, interacts with RB and regulates E2F functionOncogene, 1999
- E2F1-induced apoptosis requires DNA binding but not transactivation and is inhibited by the retinoblastoma protein through direct interaction.Genes & Development, 1997
- Apoptosis is associated with cleavage of a 5 kDa fragment from RB which mimics dephosphorylation and modulates E2F bindingOncogene, 1997
- E2F-1 Functions in Mice to Promote Apoptosis and Suppress ProliferationCell, 1996
- Tumor Induction and Tissue Atrophy in Mice Lacking E2F-1Cell, 1996
- Differential Regulation of Retinoblastoma Protein Function by Specific Cdk Phosphorylation SitesJournal of Biological Chemistry, 1996
- Cyclin A-kinase regulation of E2F-1 DNA binding function underlies suppression of an S phase checkpointCell, 1995
- Prohibitin Antiproliferative Activity and Lack of Heterozygosity in Immortalized Cell LinesExperimental Cell Research, 1995
- Stimulation of E2F1/DP1 transcriptional activity by MDM2 oncoproteinNature, 1995