Mutations of Ros Differentially Effecting Signal Transduction Pathways Leading to Cell Growth Versus Transformation
Open Access
- 1 January 1997
- journal article
- Published by Elsevier
- Vol. 272 (3) , 1500-1506
- https://doi.org/10.1074/jbc.272.3.1500
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- Phosphatidylinositol-3-OH kinase direct target of RasNature, 1994
- Identification of sequences required for the efficient localization of the focal adhesion kinase, pp125FAK, to cellular focal adhesions.The Journal of cell biology, 1993
- Phospholipase C-γ1 and phosphatidylinositol 3 kinase are the downstream mediators of the PDGF receptor's mitogenic signalCell, 1993
- SH2 and SH3 domains: From structure to functionCell, 1992
- The small GTP-binding protein rho regulates the assembly of focal adhesions and actin stress fibers in response to growth factorsCell, 1992
- PDGF stimulation of inositol phospholipid hydrolysis requires PLC-γ1 phosphorylation on tyrosine residues 783 and 1254Cell, 1991
- PDGF-Induced Activation of Phospholipase C Is Not Required for Induction of DNA SynthesisScience, 1990
- Mutation of the insulin receptor at tyrosine 960 inhibits signal transmission but does not affect its tyrosine kinase activityCell, 1988
- Common elements in growth factor stimulation and oncogenic transformation: 85 kd phosphoprotein and phosphatidylinositol kinase activityCell, 1987
- Replacement of insulin receptor tyrosine residues 1162 and 1163 compromises insulin-stimulated kinase activity and uptake of 2-deoxyglucoseCell, 1986