A neuronal model of Alzheimer's disease: An insight into the mechanisms of oxidative stress–mediated mitochondrial injury
- 7 February 2008
- journal article
- Published by Elsevier in Neuroscience
- Vol. 153 (1) , 120-130
- https://doi.org/10.1016/j.neuroscience.2008.01.044
Abstract
No abstract availableKeywords
This publication has 65 references indexed in Scilit:
- Mn porphyrin-based superoxide dismutase (SOD) mimic, MnIIITE-2-PyP5+, targets mouse heart mitochondriaFree Radical Biology & Medicine, 2007
- Specificity Protein 1-dependent p53-mediated Suppression of Human Manganese Superoxide Dismutase Gene ExpressionJournal of Biological Chemistry, 2006
- Mutations in amyloid precursor protein and presenilin‐1 genes increase the basal oxidative stress in murine neuronal cells and lead to increased sensitivity to oxidative stress mediated by amyloid β‐peptide (1–42), H2O2 and kainic acid: implications for Alzheimer's diseaseJournal of Neurochemistry, 2006
- Oxidative Stress, Accumulation of Biological 'Garbage', and AgingAntioxidants and Redox Signaling, 2006
- Manganese Superoxide Dismutase Induces p53-Dependent Senescence in Colorectal Cancer CellsMolecular and Cellular Biology, 2005
- Oxidative imbalance in patients with mild cognitive impairment and Alzheimer's diseaseNeurobiology of Aging, 2005
- Natural oligomers of the amyloid-β protein specifically disrupt cognitive functionNature Neuroscience, 2004
- Oxidative Stress Hypothesis in Alzheimer's DiseasePublished by Elsevier ,1998
- Increased Peroxidation and Reduced Antioxidant Enzyme Activity in Alzheimer's DiseaseExperimental Neurology, 1998
- Segregation of a missense mutation in the amyloid precursor protein gene with familial Alzheimer's diseaseNature, 1991