Hepatitis B Virus X Protein Differentially Regulates Cell Cycle Progression in X-transforming Versus Nontransforming Hepatocyte (AML12) Cell Lines

This publication has 93 references indexed in Scilit:

• Mitochondrially Associated Hepatitis B Virus X Protein Constitutively Activates Transcription Factors STAT-3 and NF-κB via Oxidative Stress
  Molecular and Cellular Biology, 2001
• Hepatitis B Virus X Protein Differentially Activates RAS-RAF-MAPK and JNK Pathways in X-transforming Versus Non-transforming AML12 Hepatocytes
  Journal of Biological Chemistry, 2001
• Mechanism of hyperploid cell formation induced by microtubule inhibiting drug in glioma cell lines
  Oncogene, 2001
• Role of NF-κB and Myc Proteins in Apoptosis Induced by Hepatitis B Virus HBx Protein
  Journal of Virology, 2001
• Association of p19ARF with Mdm2 inhibits ubiquitin ligase activity of Mdm2 for tumor suppressor p53
  The EMBO Journal, 1999
• Differential Immediate Early Gene Expression in Conditional Hepatitis B Virus pX-transforming VersusNontransforming Hepatocyte Cell Lines
  Published by Elsevier ,1999
• BCL-2 FAMILY: Regulators of Cell Death
  Annual Review of Immunology, 1998
• The Hepatitis B Virus X Protein Enhances the DNA Binding Potential and Transcription Efficacy of bZip Transcription Factors
  Published by Elsevier ,1997
• G1 phase progression: Cycling on cue
  Cell, 1994
• Universal control mechanism regulating onset of M-phase
  Nature, 1990