p53 Mutations are common in pancreatic cancer and are absent in chronic pancreatitis
- 14 May 1993
- journal article
- Published by Elsevier in Cancer Letters
- Vol. 69 (3) , 151-160
- https://doi.org/10.1016/0304-3835(93)90168-9
Abstract
No abstract availableKeywords
This publication has 26 references indexed in Scilit:
- Overexpression of the epidermal growth factor receptor in human pancreatic cancer is associated with concomitant increases in the levels of epidermal growth factor and transforming growth factor alpha.Journal of Clinical Investigation, 1992
- ras-Induced hyperplasia occurs with mutation of p53, but activated ras and myc together can induce carcinoma without p53 mutationCell, 1992
- p53 Mutations in Human CancersScience, 1991
- Transforming growth factor alpha and epidermal growth factor in human pancreatic cancerThe Journal of Pathology, 1991
- Patterns of expression of the p53 tumour suppressor in human breast tissues and tumours in situ and in vitroInternational Journal of Cancer, 1990
- The c‐erbB‐2 proto‐oncogene in human pancreatic cancerThe Journal of Pathology, 1990
- High frequency of Ki‐ras codon 12 mutations in pancreatic adenocarcinomasInternational Journal of Cancer, 1989
- Pathogenesis and Epidemiology of Chronic PancreatitisAnnual Review of Medicine, 1989
- Most human carcinomas of the exocrine pancreas contain mutant c-K-ras genesPublished by Elsevier ,1988
- T antigen is bound to a host protein in SY40-transformed cellsNature, 1979