Role of Tyrosine Kinase Jak2 in Prolactin-induced Differentiation and Growth of Mammary Epithelial Cells
Open Access
- 1 April 2002
- journal article
- Published by Elsevier
- Vol. 277 (16) , 14020-14030
- https://doi.org/10.1074/jbc.m112399200
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- Constitutive activation of Stat3 by the Src and JAK tyrosine kinases participates in growth regulation of human breast carcinoma cellsOncogene, 2001
- Activation and Association of the Tec Tyrosine Kinase with the Human Prolactin Receptor: Mapping of a Tec/Vav1-Receptor Binding SiteMolecular Endocrinology, 2001
- Constitutive Tyrosine Phosphorylation of ErbB-2 via Jak2 by Autocrine Secretion of Prolactin in Human Breast CancerPublished by Elsevier ,2000
- Stat3 as an OncogenePublished by Elsevier ,1999
- Differential Effects of Prolactin andsrc/abl Kinases on the Nuclear Translocation of STAT5B and STAT5APublished by Elsevier ,1999
- Activation of the prolactin receptor but not the growth hormone receptor is important for induction of mammary tumors in transgenic mice.Journal of Clinical Investigation, 1997
- Defective mammopoiesis, but normal hematopoiesis, in mice with a targeted disruption of the prolactin geneThe EMBO Journal, 1997
- Prolactin Signaling in Mammary Gland DevelopmentJournal of Biological Chemistry, 1997
- Stat5a is mandatory for adult mammary gland development and lactogenesis.Genes & Development, 1997
- Cloning of the gene encoding rat JAK2, a protein tyrosine kinaseGene, 1995