RETRACTED: Activation of the PPAR/PGC-1α Pathway Prevents a Bioenergetic Deficit and Effectively Improves a Mitochondrial Myopathy Phenotype
- 1 September 2008
- journal article
- retracted article
- Published by Elsevier in Cell Metabolism
- Vol. 8 (3) , 249-256
- https://doi.org/10.1016/j.cmet.2008.07.006
Abstract
No abstract availableKeywords
This publication has 24 references indexed in Scilit:
- Pathophysiology and fate of hepatocytes in a mouse model of mitochondrial hepatopathiesGut, 2007
- Isolated cytochrome c oxidase deficiency as a cause of MELASJournal of Medical Genetics, 2007
- Mitochondrial Diseases: Therapeutic ApproachesBioscience Reports, 2007
- PGC-1α regulates the neuromuscular junction program and ameliorates Duchenne muscular dystrophyGenes & Development, 2007
- Improved skeletal muscle oxidative enzyme activity and restoration of PGC-1α and PPARβ/δ gene expression upon rosiglitazone treatment in obese patients with type 2 diabetes mellitusInternational Journal of Obesity, 2007
- PPARδ, but not PPARα, activates PGC-1α gene transcription in muscleBiochemical and Biophysical Research Communications, 2007
- Mice lacking COX10 in skeletal muscle recapitulate the phenotype of progressive mitochondrial myopathies associated with cytochrome c oxidase deficiencyHuman Molecular Genetics, 2005
- Thiazolidinedione Activation of Peroxisome Proliferator-activated Receptor γ Can Enhance Mitochondrial Potential and Promote Cell SurvivalJournal of Biological Chemistry, 2002
- Transcriptional co-activator PGC-1α drives the formation of slow-twitch muscle fibresNature, 2002
- Mechanisms Controlling Mitochondrial Biogenesis and Respiration through the Thermogenic Coactivator PGC-1Cell, 1999