The molecular basis of a hereditary form of hypertension, glucocorticoid-remediable aldosteronism
- 31 March 1993
- journal article
- review article
- Published by Elsevier in Trends in Endocrinology & Metabolism
- Vol. 4 (2) , 57-61
- https://doi.org/10.1016/s1043-2760(05)80016-5
Abstract
No abstract availableKeywords
This publication has 13 references indexed in Scilit:
- Hereditary hypertension caused by chimaeric gene duplications and ectopic expression of aldosterone synthaseNature Genetics, 1992
- Glucocorticoid-remediable Aldosteronism in a Large Kindred: Clinical Spectrum and Diagnosis Using a Characteristic Biochemical PhenotypeAnnals of Internal Medicine, 1992
- A chimaeric llβ-hydroxylase/aldosterone synthase gene causes glucocorticoid-remediable aldosteronism and human hypertensionNature, 1992
- Defective Fasciculata Zone Function as the Mechanism of Glucocorticoid-Remediable Aldosteronism*Journal of Clinical Endocrinology & Metabolism, 1990
- Cloning and expression of a cDNA for human cytochrome P-450aldo as related to primary aldosteronismBiochemical and Biophysical Research Communications, 1990
- Glucocorticoid-suppressible hyperaldosteronism: An updateThe American Journal of Medicine, 1990
- Regulation of the Synthesis of Steroidogenic Enzymes in Adrenal Cortical Cells by ACTHAnnual Review of Physiology, 1988
- Molecular Biology of Visual PigmentsAnnual Review of Neuroscience, 1987
- Long Term Evolution of Glucocorticoid-Suppressible Hyperaldosteronism*Journal of Clinical Endocrinology & Metabolism, 1987
- ELEVATED URINARY EXCRETION OF 18-OXOCORTISOL IN GLUCOCORTICOID-SUPPRESSIBLE ALDOSTERONISMJournal of Clinical Endocrinology & Metabolism, 1984