TOXIN T46 FROM PTYCHODISCUS-BREVIS (FORMERLY GYMNODINIUM-BREVE) ENHANCES ACTIVATION OF VOLTAGE-SENSITIVE SODIUM-CHANNELS BY VERATRIDINE
- 1 January 1981
- journal article
- research article
- Vol. 19 (2) , 345-348
Abstract
No abstract availableThis publication has 9 references indexed in Scilit:
- Neurotoxin binding to receptor sites associated with voltage-sensitive sodium channels in intact, lysed, and detergent-solubilized brain membranes.Journal of Biological Chemistry, 1979
- High Pressure Liquid Chromatographic Separation of Two Major Toxic Compounds from Gymnodinium breve DavisJournal of Chromatographic Science, 1979
- Sea anemone toxin and scorpion toxin share a common receptor site associated with the action potential sodium ionophore.Journal of Biological Chemistry, 1978
- Electrophysiological and biochemical characteristics of Gymnodinium breve toxinsToxicon, 1978
- Membrane potential-dependent binding of scorpion toxin to the action potential Na+ ionophore. Studies with a toxin derivative prepared by lactoperoxidase-catalyzed iodinationJournal of Biological Chemistry, 1977
- Activation of the action potential Na+ ionophore by neurotoxins. An allosteric modelJournal of Biological Chemistry, 1977
- How Gymnodinium breve red tide toxin(s) produces repetitive firing in squid axonsAmerican Journal of Physiology-Cell Physiology, 1976
- A new method for labelling saxitoxin and its binding to non‐myelinated fibres of the rabbit vagus, lobster walking leg, and garfish olfactory nerves.The Journal of Physiology, 1976
- Purification of a toxic protein from scorpion venom which activates the action potential Na+ ionophore.Journal of Biological Chemistry, 1976