Inactivation of the zebrafish homologue of Chx10 by antisense oligonucleotides causes eye malformations similar to the ocular retardation phenotype
- 20 May 1997
- journal article
- Published by Elsevier in Mechanisms of Development
- Vol. 63 (2) , 133-143
- https://doi.org/10.1016/s0925-4773(97)00036-1
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- Ocular retardation mouse caused by Chx10 homeobox null allele: impaired retinal progenitor proliferation and bipolar cell differentiationNature Genetics, 1996
- New perspectives on eye evolutionCurrent Opinion in Genetics & Development, 1995
- Induction of Ectopic Eyes by Targeted Expression of the eyeless Gene in DrosophilaScience, 1995
- Inhibition of protein kinase C-alpha expression in mice after systemic administration of phosphorothioate antisense oligodeoxynucleotides.Proceedings of the National Academy of Sciences, 1994
- Inhibition of retinoic acid-induced activation of 3' human HOXB genes by antisense oligonucleotides affects sequential activation of genes located upstream in the four HOX clusters.Proceedings of the National Academy of Sciences, 1994
- The drosophila sine oculis locus encodes a homeodomain-containing protein required for the development of the entire visual systemNeuron, 1994
- Homeobox genes are expressed in the retina and brain of adult goldfish.Proceedings of the National Academy of Sciences, 1993
- Mouse Small eye results from mutations in a paired-like homeobox-containing geneNature, 1991
- Homeobox containing genes in the nematodeCaenorhabditis elegansNucleic Acids Research, 1990
- Point mutations define a sequence flanking the AUG initiator codon that modulates translation by eukaryotic ribosomesCell, 1986