Alzheimer's disease-like dystrophic neurites characteristically associated with senile plaques are not found within other neurodegenerative disease unless amyloid β-protein deposition is present
Open Access
- 19 March 1993
- journal article
- Published by Elsevier in Brain Research
- Vol. 606 (1) , 10-18
- https://doi.org/10.1016/0006-8993(93)91563-8
Abstract
No abstract availableKeywords
This publication has 39 references indexed in Scilit:
- Distribution of neurotensin immunoreactivity within the human amygdaloid complex: A comparison with acetylcholinesterase‐ and Nissl‐stained tissue sectionsJournal of Comparative Neurology, 1992
- Neuropathological stageing of Alzheimer-related changesActa Neuropathologica, 1991
- Catecholamine-containing axon terminals in the hypoglossal nucleus of the rat: an immuno-electronmicroscopic studyExperimental Brain Research, 1990
- Alzheimer patients: preamyloid deposits are more widely distributed than senile plaques throughout the central nervous systemNeuroscience Letters, 1989
- Substance P and somatostatin coexist within neuritic plaques: Implications for the pathogenesis of Alzheimer's diseaseNeuroscience, 1989
- Response of septal cholinergic neurons to axotomyJournal of Comparative Neurology, 1987
- Senile plaques as aberrant sprout-stimulating structuresExperimental Neurology, 1986
- Neuropeptides in neurological diseaseAnnals of Neurology, 1986
- Occurrence of neuropil threads in the senile human brain and in Alzheimer's disease: A third location of paired helical filaments outside of neurofibrillary tangles and neuritic plaquesNeuroscience Letters, 1986
- Seven peptides derived from pro-somatostatin in rat brainBiochemical and Biophysical Research Communications, 1982