The Proliferative and Migratory Activities of Breast Cancer Cells Can Be Differentially Regulated by Heparan Sulfates

This publication has 55 references indexed in Scilit:

• Growth control mechanisms in normal and transformed intestinal cells
  Philosophical Transactions Of The Royal Society B-Biological Sciences, 1998
• Fibroblast growth factors and their receptors
  Biochemistry and Cell Biology, 1997
• Heparan Sulfate Proteoglycans Play a Dual Role in Regulating Fibroblast Growth Factor-2 Mitogenic Activity in Human Breast Cancer Cells
  Experimental Cell Research, 1996
• The human diploid fibroblast senescence pathway is independent of interleukin-1α mRNA levels and tyrosine phosphorylation of FGFR-1 substrates
  Biochimica et Biophysica Acta (BBA) - Molecular Cell Research, 1996
• FGF-1-dependent proliferative and migratory responses are impaired in senescent human umbilical vein endothelial cells and correlate with the inability to signal tyrosine phosphorylation of fibroblast growth factor receptor-1 substrates.
  The Journal of cell biology, 1996
• Myc but not Fos rescue of PDGF signalling block caused by kinase-inactive Src
  Nature, 1995
• In the clutches of proteoglycans: how does heparan sulfate regulate FGF binding?
  Chemistry & Biology, 1995
• Heparan Sulfates Mediate the Binding of Basic Fibroblast Growth Factor to a Specific Receptor on Neural Precursor Cells
  Journal of Biological Chemistry, 1995
• Reduced cell motility and enhanced focal adhesion contact formation in cells from FAK-deficient mice
  Nature, 1995
• MAP kinase binds to the NH₂‐terminal activation domain of c‐Myc
  FEBS Letters, 1994