Oxidative stress and depletion of hepatic long-chain polyunsaturated fatty acids may contribute to nonalcoholic fatty liver disease
- 1 November 2004
- journal article
- review article
- Published by Elsevier in Free Radical Biology & Medicine
- Vol. 37 (9) , 1499-1507
- https://doi.org/10.1016/j.freeradbiomed.2004.06.033
Abstract
No abstract availableKeywords
This publication has 59 references indexed in Scilit:
- Polyunsaturated Fatty Acids Ameliorate Hepatic Steatosis in Obese Mice by Srebp–1 SuppressionHepatology, 2003
- POLYENYLPHOSPHATIDYLCHOLINE CORRECTS THE ALCOHOL-INDUCED HEPATIC OXIDATIVE STRESS BY RESTORING S-ADENOSYLMETHIONINEAlcohol and Alcoholism, 2003
- Cellular response to oxidative stress: Signaling for suicide and survival*Journal of Cellular Physiology, 2002
- Mechanisms of HepatotoxicityToxicological Sciences, 2002
- Delivery of IκB superrepressor gene with adenovirus reduces early alcohol-induced liver injury in ratsHepatology, 2001
- Non-alcoholic fatty liver disease in an area of southern Italy: main clinical, histological, and pathophysiological aspectsJournal of Hepatology, 2001
- Nonalcoholic fatty liver disease: A spectrum of clinical and pathological severity☆, ☆☆Gastroenterology, 1999
- Peroxisome proliferator–activated receptor α mediates the adaptive response to fastingJournal of Clinical Investigation, 1999
- Animal model of systemic carnitine deficiency: Analysis in C3H-H-2° strain of mouse associated with juvenile visceral steatosisBiochemical and Biophysical Research Communications, 1991
- Biochemistry of Oxidative StressAngewandte Chemie International Edition in English, 1986