Transgenic Mice Over-Expressing the C-99 Fragment of βPP with an α-Secretase Site Mutation Develop a Myopathy Similar to Human Inclusion Body Myositis
- 1 December 1998
- journal article
- Published by Elsevier in The American Journal of Pathology
- Vol. 153 (6) , 1679-1686
- https://doi.org/10.1016/s0002-9440(10)65681-7
Abstract
No abstract availableKeywords
Funding Information
- National Institute on Aging (P30AG13280, P50AG05136, T32AG00057, R35AG10917)
This publication has 44 references indexed in Scilit:
- Apolipoprotein E and apolipoprotein E messenger RNA in muscle of inclusion body myositis and myopathiesAnnals of Neurology, 1996
- Apolipoprotein E immunoreactive deposits in inclusion-body muscle diseasesThe Lancet, 1994
- The Toxic Effect of Alzheimer Amyloid Protein Precursor Overexpressed in the Neuroblastoma Cell Line NB-1 on Neurite OutgrowthGerontology, 1994
- Prion protein is abnormally accumulated in inclusion-body myositisNeuroReport, 1993
- Degeneration in vitro of post-mitotic neurons overexpressing the Alzheimer amyloid protein precursorNature, 1992
- Potentially Amyloidogenic, Carboxyl-Terminal Derivatives of the Amyloid Protein PrecursorScience, 1992
- Overexpression of amyloid precursor protein alters its normal processing and is associated with neurotoxicityBiochemical and Biophysical Research Communications, 1992
- Amyloid deposition as the central event in the aetiology of Alzheimer's diseaseTrends in Pharmacological Sciences, 1991
- Cleavage of Amyloid β Peptide During Constitutive Processing of Its PrecursorScience, 1990
- Use of avidin-biotin-peroxidase complex (ABC) in immunoperoxidase techniques: a comparison between ABC and unlabeled antibody (PAP) procedures.Journal of Histochemistry & Cytochemistry, 1981