Antioxidant Properties of Pyruvate Mediate its Potentiation of β -Adrenergic Inotropism in Stunned Myocardium

This publication has 30 references indexed in Scilit:

• The Cumulative Nature of Pyruvate's Dual Mechanism for Myocardial Protection
  Journal of Surgical Research, 1995
• Protection by pyrdvate against inhibition of Na+,K+-ATPase by a free radical generating system containing T-butylhydroperoxide
  Life Sciences, 1995
• The metabolic consequences of hydroperoxide perfusion on the isolated rat heart
  European Journal of Biochemistry, 1989
• Marked reduction of free radical generation and contractile dysfunction by antioxidant therapy begun at the time of reperfusion. Evidence that myocardial "stunning" is a manifestation of reperfusion injury.
  Circulation Research, 1989
• Pyruvate‐enhanced phosphorylation potential and inotropism in normoxic and postischemic isolated working heart
  European Journal of Biochemistry, 1989
• Reversal of dysfunction in postischemic stunned myocardium by epinephrine and postextrasystolic potentiation
  Journal of the American College of Cardiology, 1986
• Nonenzymatic decarboxylation of pyruvate
  Analytical Biochemistry, 1984
• Response of reperfusion-salvaged, stunned myocardium to inotropic stimulation
  American Heart Journal, 1984
• Inactivation of cardiac adenylate cyclase by oxidation, trivalent arsenicals, and N-ethylmaleimide
  Archives of Biochemistry and Biophysics, 1981
• Properties of the citrate transporter in rat heart: implications for regulation of glycolysis by cytosolic citrate
  Canadian Journal of Biochemistry, 1976