Lethal weapon: amyloid β-peptide, role in the oxidative stress and neurodegeneration of Alzheimer’s disease
- 30 June 2004
- journal article
- review article
- Published by Elsevier in Neurobiology of Aging
- Vol. 25 (5) , 581-587
- https://doi.org/10.1016/j.neurobiolaging.2004.02.002
Abstract
No abstract availableThis publication has 46 references indexed in Scilit:
- Alzheimer’s amyloid β-peptide (1–42): involvement of methionine residue 35 in the oxidative stress and neurotoxicity properties of this peptideNeurobiology of Aging, 2004
- Neurotoxic, Redox-competent Alzheimer's β-Amyloid Is Released from Lipid Membrane by Methionine OxidationJournal of Biological Chemistry, 2003
- Selectively Reduced Expression of Synaptic Plasticity-Related Genes in Amyloid Precursor Protein + Presenilin-1 Transgenic MiceJournal of Neuroscience, 2003
- Metal Binding and Oxidation of Amyloid-β within Isolated Senile Plaque Cores: Raman Microscopic EvidenceBiochemistry, 2003
- Amyloid β-protein (Aβ) assembly: Aβ40 and Aβ42 oligomerize through distinct pathwaysProceedings of the National Academy of Sciences, 2002
- Evidence that amyloid beta-peptide-induced lipid peroxidation and its sequelae in Alzheimer’s disease brain contribute to neuronal death1Neurobiology of Aging, 2002
- Immunization reverses memory deficits without reducing brain Aβ burden in Alzheimer's disease modelNature Neuroscience, 2002
- Postsynaptic Scaffolds of Excitatory and Inhibitory Synapses in Hippocampal Neurons: Maintenance of Core Components Independent of Actin Filaments and MicrotubulesJournal of Neuroscience, 2000
- Decrease in Peptide Methionine Sulfoxide Reductase in Alzheimer's Disease BrainJournal of Neurochemistry, 1999
- Principles that Govern the Folding of Protein ChainsScience, 1973