FLT3/ITD Mutation Signaling Includes Suppression of SHP-1
Open Access
- 1 February 2005
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 280 (7) , 5361-5369
- https://doi.org/10.1074/jbc.m411974200
Abstract
No abstract availableKeywords
This publication has 64 references indexed in Scilit:
- Evidence for a positive role of SHIP in the BCR-ABL–mediated transformation of primitive murine hematopoietic cells and in human chronic myeloid leukemiaBlood, 2003
- Blocking the Function of Tyrosine Phosphatase SHP-2 by Targeting Its Src Homology 2 DomainsJournal of Biological Chemistry, 2003
- FLT3: ITDoes matter in leukemiaLeukemia, 2003
- Molecular mechanisms of transformation by the BCR-ABL oncogeneSeminars in Hematology, 2003
- Abnormal splicing of SHP-1 protein tyrosine phosphatase in human T cells: Implications for lymphomagenesisExperimental Hematology, 2003
- Targeted inhibition of FLT3 overcomes the block to myeloid differentiation in 32Dcl3 cells caused by expression of FLT3/ITD mutationsBlood, 2002
- Methyltransferase Recruitment and DNA Hypermethylation of Target Promoters by an Oncogenic Transcription FactorScience, 2002
- The SH2 domain-containing protein tyrosine phosphatase SHP-1 is induced by granulocyte colony-stimulating factor (G-CSF) and modulates signaling from the G-CSF receptorLeukemia, 2000
- Gain-of-Function Mutations of c- kit in Human Gastrointestinal Stromal TumorsScience, 1998
- SH2-Containing Phosphotyrosine Phosphatase as a Target of Protein-Tyrosine KinasesScience, 1993