E2F transcriptional repressor complexes are critical downstream targets of p19ARF/p53-induced proliferative arrest

This publication has 79 references indexed in Scilit:

• Activation of a cAMP Pathway and Induction of Melanogenesis Correlate with Association of p16INK4 and p27KIP1 to CDKs, Loss of E2F-Binding Activity, and Premature Senescence of Human Melanocytes
  Experimental Cell Research, 1999
• E2F-6: a novel member of the E2F family is an inhibitor of E2F-dependent transcription
  Oncogene, 1998
• Oncogenic ras Provokes Premature Cell Senescence Associated with Accumulation of p53 and p16INK4a
  Cell, 1997
• Analysis of Chromatin Structure byin VivoFormaldehyde Cross-Linking
  Methods, 1997
• Cyclin A-kinase regulation of E2F-1 DNA binding function underlies suppression of an S phase checkpoint
  Cell, 1995
• Regulation of the retinoblastoma protein-related p107 by G1 cyclin complexes.
  Genes & Development, 1995
• Tumour-derived p16 alleles encoding proteins defective in cell-cycle inhibition
  Nature, 1995
• Negative regulation of the growth-promoting transcription factor E2F-1 by a stably bound cyclin A-dependent protein kinase
  Cell, 1994
• A C-terminal protein-binding domain in the retinoblastoma protein regulates nuclear c-Abl tyrosine kinase in the cell cycle
  Cell, 1993
• Failure to Phosphorylate the Retinoblastoma Gene Product in Senescent Human Fibroblasts
  Science, 1990