Mutations in the UL53 gene of HSV-1 abolish virus neurovirulence to mice by the intracerebral route of infection
- 12 November 1992
- journal article
- Published by Elsevier in Virus Research
- Vol. 26 (2) , 99-112
- https://doi.org/10.1016/0168-1702(92)90150-8
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- The RL neurovirulence locus in herpes simplex virus type 2 strain HG52 plays no role in latencyJournal of General Virology, 1991
- The Complete DNA Sequence of the Long Unique Region in the Genome of Herpes Simplex Virus Type 1Journal of General Virology, 1988
- HSV-1 virulence for mice by the intracerebral route is encoded by the BamHI-L DNA fragment containing the cell fusion geneArchiv für die gesamte Virusforschung, 1987
- Replacement of the deletion in the genome (0.762–0.789 mu) of avirulent HSV-1 HFEM using cloned Mlul DNA fragment (0.7615–0.796 mu) of virulent HSV-1 F leads to generation of virulent intratypic recombinantVirus Research, 1986
- Neurovirulence of herpes simplex virus type 1 depends on age in mice and thymidine kinase expressionArchiv für die gesamte Virusforschung, 1983
- A low thymidine kinase-producing mutant of herpes simplex virus type 1 causes latent trigeminal ganglia infections in miceArchiv für die gesamte Virusforschung, 1983
- A simple method for displaying the hydropathic character of a proteinJournal of Molecular Biology, 1982
- Isolation of biologically active ribonucleic acid from sources enriched in ribonucleaseBiochemistry, 1979
- The pathogenicity of thymidine kinase-deficient mutants of herpes simplex virus in miceEpidemiology and Infection, 1978
- A general method applicable to the search for similarities in the amino acid sequence of two proteinsJournal of Molecular Biology, 1970