Molecular mechanisms associated with leptin resistance: n-3 polyunsaturated fatty acids induce alterations in the tight junction of the brain
- 1 May 2005
- journal article
- research article
- Published by Elsevier in Cell Metabolism
- Vol. 1 (5) , 331-341
- https://doi.org/10.1016/j.cmet.2005.04.004
Abstract
No abstract availableKeywords
This publication has 60 references indexed in Scilit:
- The soluble leptin receptor is crucial for leptin action: evidence from clinical and experimental dataInternational Journal of Obesity, 2003
- Two defects contribute to hypothalamic leptin resistance in mice with diet-induced obesityJournal of Clinical Investigation, 2000
- Fasting, but not adrenalectomy, reduces transport of leptin into the brain☆Peptides, 2000
- Identification of SOCS-3 as a Potential Mediator of Central Leptin ResistanceMolecular Cell, 1998
- Regulation of Tight Junction Permeability and Occludin Expression by Polyunsaturated Fatty AcidsBiochemical and Biophysical Research Communications, 1998
- Identification of targets of leptin action in rat hypothalamus.Journal of Clinical Investigation, 1996
- Functional dissociation of paracellular permeability and transepithelial electrical resistance and disruption of the apical-basolateral intramembrane diffusion barrier by expression of a mutant tight junction membrane protein.The Journal of cell biology, 1996
- Extracellular Hypothalamic Monoamines Measured by In Vivo Microdialysis in a Rat Model of Dietary Fat‐Induced ObesityObesity Research, 1994
- Occludin: a novel integral membrane protein localizing at tight junctions.The Journal of cell biology, 1993
- Breaking through the tight junction barrier.The Journal of cell biology, 1993